CELL SIGNALING TECHNOLOGY search Cell Signaling Technology MAP2K4 products
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| wb | | Cell Signaling Technology anti-phospho-MAPK kinase 4 antibody was used in western blot to study the expression of hypertonicity-induced aquaporin-1 (AQP1) which is mediated by the activation of MAPK pathways and hypertonicity-responsive element in the AQP1 gene. |
| wb | | Cell Signaling anti-MKK4 and anti-phospho-MKK4 antibodies were used in western blot to study the interaction of Sef with TAK1 which mediates JNK activation and apoptosis. |
| wb | | Cell Signaling phospho-SEK1/MKK4 antibody was used in western blot to study the inhibition of activator protein-1, NF-{kappa}B, and MAPKs and induction of phase 2 detoxifying enzyme activity by chlorogenic acid. |
SANTA CRUZ BIOTECHNOLOGY search Santa Cruz Biotechnology MAP2K4 products
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| ip, wb | | Santa Cruz Biotechnology polyclonal anti-MKK4 antibody MEK-4 H-98 was used in western blot and immunoprecipitation to study the function of tumor suppressor CYLD in the regulation of JNK signaling. |
| wb | | Santa Cruz anti-MKK4/JNKK1 antibody was used in human Jurkat cells and in western blot to study the roles for receptor-interacting protein and TRAF2 in oxidative stress-induced cell death. |
| wb | | SCBT MKK4 antibody was used in western blot to study the inhibition of activator protein-1, NF-{kappa}B, and MAPKs and induction of phase 2 detoxifying enzyme activity by chlorogenic acid. |
MILLIPORE search Millipore MAP2K4 products
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| ic, wb | | Upstate Biotechnology antibody against MKK4 was used in western blot and immunocytochemistry to study the regulation of c-Jun N-terminal kinase by MEKK-2 and mitogen-activated protein kinase kinase kinases (MAP3Ks) in rheumatoid arthritis. |
STRESSGEN search Stressgen MAP2K4 products
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| wb | | StressGen anti-MAPK kinase 4 antibody was used in western blot to study the expression of hypertonicity-induced aquaporin-1 (AQP1) which is mediated by the activation of MAPK pathways and hypertonicity-responsive element in the AQP1 gene. |
Articles Reviewed |
| 1. Fuminori Umenishi et al. Hypertonicity-induced aquaporin-1 (AQP1) expression is mediated by the activation of MAPK pathways and hypertonicity-responsive element in the AQP1 gene. 2003 |
| 2. Deepa R Hammaker et al. Regulation of c-Jun N-terminal kinase by MEKK-2 and mitogen-activated protein kinase kinase kinases in rheumatoid arthritis. 2004 |
| 3. Han-Ming Shen et al. Essential roles of receptor-interacting protein and TRAF2 in oxidative stress-induced cell death. 2004 |
| 4. Xuehui Yang et al. Sef interacts with TAK1 and mediates JNK activation and apoptosis. 2004 |
| 5. William Reiley et al. Negative regulation of JNK signaling by the tumor suppressor CYLD. 2004 |
| 6. Rentian Feng et al. Inhibition of activator protein-1, NF-kappaB, and MAPKs and induction of phase 2 detoxifying enzyme activity by chlorogenic acid. 2005 |