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BAD antibody review
BAD antibodies
This is a review about BAD antibodies, based on 24 published articles using BAD antibodies in western blot, immunohistochemistry, immunoprecipitation, immunocytochemistry, and other immunological technologies. It is aimed to help ExactAntigen visitors find the most suited BAD antibody. Information in this review (with links to publications) can be searched freely.
immunocytochemistry (ic), immunohistochemistry (ih), immunoprecipitation (ip), western blot (wb)
CELL SIGNALING TECHNOLOGY    search Cell Signaling Technology BAD products
includes other brands or spellings
ic, ih, wb    Cell Signaling Technology rabbit polyclonal anti-Bad antibody was used in western blot, immunohistochemistry and immunocytochemistry to study that overexpression of copper/zinc superoxide dismutase in transgenic mice protects against neuronal cell death after transient focal ischemia by blocking activation of the Bad cell death signaling pathway.
ip, wb    Cell Signaling Technology anti-Bad antibody was used in western blot and immunoprecipitation to study that kallikrein/kinin protects against myocardial apoptosis after ischemia/reperfusion via akt-glycogen synthase kinase-3 and akt-bad·14-3-3 signaling pathways.
ip, wb    Cell Signaling antibodies against Bad and its phosphorylated form were used in western blot to investigate the regulation PAK-2-dependent activation of LIMK by A2M in 1-LN prostate cancer cells.
ip, wb    Cell Signaling antibodies against BAD, BAD phospho-Ser-112, -Ser-136, and -Ser-155 were used in western blot/or immunoprecipitation to study BAD dissociation from 14-3-3{zeta} at each G2/M phase of proliferating lymphoid cells.
wb    Cell Signaling anti-phospho-BAD (Ser112, Ser136, Ser155) antibody was used in western blot to study that PIM-2 kinase phosphorylates BAD on Serine 112 and reverses BAD-induced cell death.
wb    Cell Signaling anti-Bad and anti-phospho-Bad (Ser136) antibodies were used in western blot to study the effect of adrenomedullin on myocardial infarction and apoptosis after ischemia and reperfusion.
wb    Cell Signaling Technology anti-human phospho-Ser136-BAD polyclonal antibody was used in western blot to detect phospho-Ser136-BAD in LNCaP and PC-3 cells.
wb    Cell Signaling Technology anti-phospho-Ser-136 BAD antibody was used in western blot to detect phospho-Ser-136 BAD in P388D1 cells stably expressing transfected myr-AKT.
wb    Cell Signaling anti-phospho-BAD antibody was used in western blot to detect phospho-BAD in HT-29 cells treated with NMT1-1 siRNA or NMT2-4 siRNA.
wb    Cell Signaling Technology polyclonal anti-Bad, polyclonal anti-phospho-Bad (Ser-136) antibodies were used in western blot to study the role for PKB and megalin in the survival or death of renal proximal tubule cells.
wb    Cell Signaling anti-phospho-Ser-136 BAD antibody 9295, anti-BAD antibody 9292 were used in human 293 cells and in western blot to study the effect of peptide spanning the A strand of the proto-oncogene TCL1 on Akt kinase activity.
wb    Cell Signaling antibody against BAD and phosphorylated BAD (Ser-112) was used in western blot to study the critical role of the mTOR-dependent suppression of protein phosphatase 2A for phospholipase D survival signals in human breast cancer cells.
wb     Cell Signaling Technologies anti-BAD (Ser-136) antibody was used in western blot to study Akt1 activation.
wb    Cell Signaling Technology anti-Bad antibody was used in western blot to study neuroprotective role of PRAS and NGF in apoptotic neuronal cell death after stroke.
wb    Cell Signaling Technology anti-phospho-Bad (Ser112 or Ser155) and anti-Bad antibodies were used in western blot to study cell survival by GLP-2R signaling.
wb    Cell Signaling Technology rabbit polyclonal phospho-BAD (Ser (P) -112 or Ser (P) -136) antibodies were used in western blot to study the effect on BAD phosphorylation and Bcl-2 association by Raf-1 phosphorylation.
SANTA CRUZ BIOTECHNOLOGY    search Santa Cruz Biotechnology BAD products
includes other brands or spellings
ih    Santa Cruz Biotech BAD antibody was tested in immunohistochemistry by HPA (CAB004205) .
wb    Santa Cruz Biotechnology goat anti-BAD antibody was used in western blot to study that PIM-2 kinase phosphorylates BAD on Serine 112 and reverses BAD-induced cell death.
wb    Santa Cruz Biotechnology rabbit anti-human BAD antibody was used in western blot to detect BAD in LNCaP and PC-3 cells.
wb    Santa Cruz Biotechnology anti-actin antibody was used in western blot to detect phosphorylation and oligomerization of Bad in U373 and PC12 cells infected with LacZ or p75NTR recombinant adenovirus.
wb    Santa Cruz Biotechnology rabbit polyclonal anti-Bad and phospho-Bad antibody was used in western blot to detect Bad and phospho-Bad in human corneal epithelial (HCE) stimulated with HGF.
wb    Santa Cruz Biotechnology polyclonal rabbit anti-human BAD and phosphorylated BAD (P-BAD) antibodies were used in human Kaposi's sarcoma cells (KS cells) and in western blot to study the molecular mechanisms through which HIV-1-Tat protein inhibits vincristine-induced apoptosis of KS cells.
wb    Santa Cruz Biotechnology Bad antibody was used in western blot to study the role of Cyr61 in endometrial tumorigenesis.
wb    Santa Cruz Biotechnology mouse monoclonal anti-Bad antibody (C-7) was used in western blot to study the role of Bid in the cell killing that occurs subsequent to activation of the Fas receptor.
BD BIOSCIENCES    search BD Biosciences BAD products
includes other brands or spellings
wb    BD Transduction Bad antibody was tested in western blot by Abminer.
wb    BD Transduction Laboratories mouse monoclonal anti-BAD antibody was used in western blot to study that PIM-2 kinase phosphorylates BAD on Serine 112 and reverses BAD-induced cell death.
wb    BD Biosciences monoclonal mouse anti-Bad antibody was used in western blot to study dendritic cell apoptosis in the maintenance of immune tolerance.


Articles Reviewed
1. Marco Tafani et al. Cytochrome c release upon Fas receptor activation depends on translocation of full-length bid and the induction of the mitochondrial permeability transition. 2002
2. Bernardo Yusta et al. Glucagon-like peptide-2 receptor activation engages bad and glycogen synthase kinase-3 in a protein kinase A-dependent manner and prevents apoptosis following inhibition of phosphatidylinositol 3-kinase. 2002
3. Maria Chiara Deregibus et al. HIV-1-Tat protein activates phosphatidylinositol 3-kinase/ AKT-dependent survival pathways in Kaposi's sarcoma cells. 2002
4. Atsushi Saito et al. Overexpression of copper/zinc superoxide dismutase in transgenic mice protects against neuronal cell death after transient focal ischemia by blocking activation of the Bad cell death signaling pathway. 2003
5. Chih-Cheng Yang et al. Bcl-xL mediates a survival mechanism independent of the phosphoinositide 3-kinase/Akt pathway in prostate cancer cells. 2003
6. Albert H Kim et al. JNK-interacting protein 1 promotes Akt1 activation. 2003
7. Kazuo Kato et al. Adrenomedullin gene delivery attenuates myocardial infarction and apoptosis after ischemia and reperfusion. 2003
8. Bin Yan et al. The PIM-2 kinase phosphorylates BAD on serine 112 and reverses BAD-induced cell death. 2003
9. Antonio E Rusiñol et al. AKT/protein kinase B regulation of BCL family members during oxysterol-induced apoptosis. 2004
10. Asha L Bhakar et al. Apoptosis induced by p75NTR overexpression requires Jun kinase-dependent phosphorylation of Bad. 2003
11. Atsushi Saito et al. Neuroprotective role of a proline-rich Akt substrate in apoptotic neuronal cell death after stroke: relationships with nerve growth factor. 2004
12. Azucena Kakazu et al. HGF protects corneal epithelial cells from apoptosis by the PI-3K/Akt-1/Bad- but not the ERK1/2-mediated signaling pathway. 2004
13. Makoto Hiromura et al. Inhibition of Akt kinase activity by a peptide spanning the betaA strand of the proto-oncogene TCL1. 2004
14. Wenwen Chien et al. Cyr61 suppresses growth of human endometrial cancer cells. 2004
15. Hang Yin et al. Kallikrein/kinin protects against myocardial apoptosis after ischemia/reperfusion via Akt-glycogen synthase kinase-3 and Akt-Bad.14-3-3 signaling pathways. 2005
16. Shenghao Jin et al. p21-activated Kinase 1 (Pak1)-dependent phosphorylation of Raf-1 regulates its mitochondrial localization, phosphorylation of BAD, and Bcl-2 association. 2005
17. Akiko Hashimoto et al. BAD detects coincidence of G2/M phase and growth factor deprivation to regulate apoptosis. 2005
18. Uma Kant Misra et al. Binding of activated alpha2-macroglobulin to its cell surface receptor GRP78 in 1-LN prostate cancer cells regulates PAK-2-dependent activation of LIMK. 2005
19. Li Hui et al. mTOR-dependent suppression of protein phosphatase 2A is critical for phospholipase D survival signals in human breast cancer cells. 2005
20. Charles E Ducker et al. Two N-myristoyltransferase isozymes play unique roles in protein myristoylation, proliferation, and apoptosis. 2005
21. Min Chen et al. Dendritic cell apoptosis in the maintenance of immune tolerance. 2006
22. Sylvia M Major et al. AbMiner: a bioinformatic resource on available monoclonal antibodies and corresponding gene identifiers for genomic, proteomic, and immunologic studies. 2006
23. Anja Persson et al. A human protein atlas based on antibody proteomics. 2006
24. Celso Caruso-Neves et al. PKB and megalin determine the survival or death of renal proximal tubule cells. 2006


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