CELL SIGNALING TECHNOLOGY search Cell Signaling Technology BAD products includes other brands or spellings
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| ic, ih, wb | | Cell Signaling Technology rabbit polyclonal anti-Bad antibody was used in western blot, immunohistochemistry and immunocytochemistry to study that overexpression of copper/zinc superoxide dismutase in transgenic mice protects against neuronal cell death after transient focal ischemia by blocking activation of the Bad cell death signaling pathway. |
| ip, wb | | Cell Signaling Technology anti-Bad antibody was used in western blot and immunoprecipitation to study that kallikrein/kinin protects against myocardial apoptosis after ischemia/reperfusion via akt-glycogen synthase kinase-3 and akt-bad·14-3-3 signaling pathways. |
| ip, wb | | Cell Signaling antibodies against Bad and its phosphorylated form were used in western blot to investigate the regulation PAK-2-dependent activation of LIMK by A2M in 1-LN prostate cancer cells. |
| ip, wb | | Cell Signaling antibodies against BAD, BAD phospho-Ser-112, -Ser-136, and -Ser-155 were used in western blot/or immunoprecipitation to study BAD dissociation from 14-3-3{zeta} at each G2/M phase of proliferating lymphoid cells. |
| wb | | Cell Signaling anti-phospho-BAD (Ser112, Ser136, Ser155) antibody was used in western blot to study that PIM-2 kinase phosphorylates BAD on Serine 112 and reverses BAD-induced cell death. |
| wb | | Cell Signaling anti-Bad and anti-phospho-Bad (Ser136) antibodies were used in western blot to study the effect of adrenomedullin on myocardial infarction and apoptosis after ischemia and reperfusion. |
| wb | | Cell Signaling Technology anti-human phospho-Ser136-BAD polyclonal antibody was used in western blot to detect phospho-Ser136-BAD in LNCaP and PC-3 cells. |
| wb | | Cell Signaling Technology anti-phospho-Ser-136 BAD antibody was used in western blot to detect phospho-Ser-136 BAD in P388D1 cells stably expressing transfected myr-AKT. |
| wb | | Cell Signaling anti-phospho-BAD antibody was used in western blot to detect phospho-BAD in HT-29 cells treated with NMT1-1 siRNA or NMT2-4 siRNA. |
| wb | | Cell Signaling Technology polyclonal anti-Bad, polyclonal anti-phospho-Bad (Ser-136) antibodies were used in western blot to study the role for PKB and megalin in the survival or death of renal proximal tubule cells. |
| wb | | Cell Signaling anti-phospho-Ser-136 BAD antibody 9295, anti-BAD antibody 9292 were used in human 293 cells and in western blot to study the effect of peptide spanning the A strand of the proto-oncogene TCL1 on Akt kinase activity. |
| wb | | Cell Signaling antibody against BAD and phosphorylated BAD (Ser-112) was used in western blot to study the critical role of the mTOR-dependent suppression of protein phosphatase 2A for phospholipase D survival signals in human breast cancer cells. |
| wb | | Cell Signaling Technologies anti-BAD (Ser-136) antibody was used in western blot to study Akt1 activation. |
| wb | | Cell Signaling Technology anti-Bad antibody was used in western blot to study neuroprotective role of PRAS and NGF in apoptotic neuronal cell death after stroke. |
| wb | | Cell Signaling Technology anti-phospho-Bad (Ser112 or Ser155) and anti-Bad antibodies were used in western blot to study cell survival by GLP-2R signaling. |
| wb | | Cell Signaling Technology rabbit polyclonal phospho-BAD (Ser (P) -112 or Ser (P) -136) antibodies were used in western blot to study the effect on BAD phosphorylation and Bcl-2 association by Raf-1 phosphorylation. |
SANTA CRUZ BIOTECHNOLOGY search Santa Cruz Biotechnology BAD products includes other brands or spellings
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| ih | | Santa Cruz Biotech BAD antibody was tested in immunohistochemistry by HPA (CAB004205) . |
| wb | | Santa Cruz Biotechnology goat anti-BAD antibody was used in western blot to study that PIM-2 kinase phosphorylates BAD on Serine 112 and reverses BAD-induced cell death. |
| wb | | Santa Cruz Biotechnology rabbit anti-human BAD antibody was used in western blot to detect BAD in LNCaP and PC-3 cells. |
| wb | | Santa Cruz Biotechnology anti-actin antibody was used in western blot to detect phosphorylation and oligomerization of Bad in U373 and PC12 cells infected with LacZ or p75NTR recombinant adenovirus. |
| wb | | Santa Cruz Biotechnology rabbit polyclonal anti-Bad and phospho-Bad antibody was used in western blot to detect Bad and phospho-Bad in human corneal epithelial (HCE) stimulated with HGF. |
| wb | | Santa Cruz Biotechnology polyclonal rabbit anti-human BAD and phosphorylated BAD (P-BAD) antibodies were used in human Kaposi's sarcoma cells (KS cells) and in western blot to study the molecular mechanisms through which HIV-1-Tat protein inhibits vincristine-induced apoptosis of KS cells. |
| wb | | Santa Cruz Biotechnology Bad antibody was used in western blot to study the role of Cyr61 in endometrial tumorigenesis. |
| wb | | Santa Cruz Biotechnology mouse monoclonal anti-Bad antibody (C-7) was used in western blot to study the role of Bid in the cell killing that occurs subsequent to activation of the Fas receptor. |
BD BIOSCIENCES search BD Biosciences BAD products includes other brands or spellings
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| wb | | BD Transduction Bad antibody was tested in western blot by Abminer. |
| wb | | BD Transduction Laboratories mouse monoclonal anti-BAD antibody was used in western blot to study that PIM-2 kinase phosphorylates BAD on Serine 112 and reverses BAD-induced cell death. |
| wb | | BD Biosciences monoclonal mouse anti-Bad antibody was used in western blot to study dendritic cell apoptosis in the maintenance of immune tolerance. |
Articles Reviewed |
| 1. Marco Tafani et al. Cytochrome c release upon Fas receptor activation depends on translocation of full-length bid and the induction of the mitochondrial permeability transition. 2002 |
| 2. Bernardo Yusta et al. Glucagon-like peptide-2 receptor activation engages bad and glycogen synthase kinase-3 in a protein kinase A-dependent manner and prevents apoptosis following inhibition of phosphatidylinositol 3-kinase. 2002 |
| 3. Maria Chiara Deregibus et al. HIV-1-Tat protein activates phosphatidylinositol 3-kinase/ AKT-dependent survival pathways in Kaposi's sarcoma cells. 2002 |
| 4. Atsushi Saito et al. Overexpression of copper/zinc superoxide dismutase in transgenic mice protects against neuronal cell death after transient focal ischemia by blocking activation of the Bad cell death signaling pathway. 2003 |
| 5. Chih-Cheng Yang et al. Bcl-xL mediates a survival mechanism independent of the phosphoinositide 3-kinase/Akt pathway in prostate cancer cells. 2003 |
| 6. Albert H Kim et al. JNK-interacting protein 1 promotes Akt1 activation. 2003 |
| 7. Kazuo Kato et al. Adrenomedullin gene delivery attenuates myocardial infarction and apoptosis after ischemia and reperfusion. 2003 |
| 8. Bin Yan et al. The PIM-2 kinase phosphorylates BAD on serine 112 and reverses BAD-induced cell death. 2003 |
| 9. Antonio E Rusiñol et al. AKT/protein kinase B regulation of BCL family members during oxysterol-induced apoptosis. 2004 |
| 10. Asha L Bhakar et al. Apoptosis induced by p75NTR overexpression requires Jun kinase-dependent phosphorylation of Bad. 2003 |
| 11. Atsushi Saito et al. Neuroprotective role of a proline-rich Akt substrate in apoptotic neuronal cell death after stroke: relationships with nerve growth factor. 2004 |
| 12. Azucena Kakazu et al. HGF protects corneal epithelial cells from apoptosis by the PI-3K/Akt-1/Bad- but not the ERK1/2-mediated signaling pathway. 2004 |
| 13. Makoto Hiromura et al. Inhibition of Akt kinase activity by a peptide spanning the betaA strand of the proto-oncogene TCL1. 2004 |
| 14. Wenwen Chien et al. Cyr61 suppresses growth of human endometrial cancer cells. 2004 |
| 15. Hang Yin et al. Kallikrein/kinin protects against myocardial apoptosis after ischemia/reperfusion via Akt-glycogen synthase kinase-3 and Akt-Bad.14-3-3 signaling pathways. 2005 |
| 16. Shenghao Jin et al. p21-activated Kinase 1 (Pak1)-dependent phosphorylation of Raf-1 regulates its mitochondrial localization, phosphorylation of BAD, and Bcl-2 association. 2005 |
| 17. Akiko Hashimoto et al. BAD detects coincidence of G2/M phase and growth factor deprivation to regulate apoptosis. 2005 |
| 18. Uma Kant Misra et al. Binding of activated alpha2-macroglobulin to its cell surface receptor GRP78 in 1-LN prostate cancer cells regulates PAK-2-dependent activation of LIMK. 2005 |
| 19. Li Hui et al. mTOR-dependent suppression of protein phosphatase 2A is critical for phospholipase D survival signals in human breast cancer cells. 2005 |
| 20. Charles E Ducker et al. Two N-myristoyltransferase isozymes play unique roles in protein myristoylation, proliferation, and apoptosis. 2005 |
| 21. Min Chen et al. Dendritic cell apoptosis in the maintenance of immune tolerance. 2006 |
| 22. Sylvia M Major et al. AbMiner: a bioinformatic resource on available monoclonal antibodies and corresponding gene identifiers for genomic, proteomic, and immunologic studies. 2006 |
| 23. Anja Persson et al. A human protein atlas based on antibody proteomics. 2006 |
| 24. Celso Caruso-Neves et al. PKB and megalin determine the survival or death of renal proximal tubule cells. 2006 |