ExactAntigen

Rabbit Polyclonal anti-PRAS40
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CatalogCode:NB200-559
ProductName:PRAS40 Antibody
Product Description:Rabbit Polyclonal anti-PRAS40
Clonality:Polyclonal
Immunogen:This antibody was produced against a chemically synthesized phosphopeptide derived from the region of PRAS40 that contains threonine 246 conjugated to Alexa Fluor
CrossReactivity:Cross-reacts with Human and Mouse. Not yet tested in other species.
Packaging:0.1 ml Rabbit antisera.
Uses:Not tested in other applications. Optimal dilutions/concentrations should be determined by the end user.
Localization:Nuclear. Central region of the nuclear pore, within the transporter. During mitoticcell division, it associates with the poles of the mitotic spindle.
Control:NIH3T3 cells.
Background:Proline-Rich Akt Substrate (PRAS40) is a 40 kDa substrate of Akt. Akt and PRAS40 can be found in the phosphoinositide 3-kinase (PI3K) pathway, which plays a role in glucose uptake, cell growth, and apoptosis inhibition. PRAS40 is a 14-3-3 binding protein that reacts with insulin, but whose precise function is not yet known. It has been hypothesized that it may play a role in interacting with SH3 and WW domain containing proteins. The binding of PRAS40 to these proteins may bring about a change of function. Activated Akt phosphorylates PRAS40 on threonine 246, which is thereby able to bind 14-3-3. Mutation of PRAS40 threonine 246 has been shown to be apoptotic, and plays a role in abrogating neuronal protection.
Storage:Store at 4C short term. Aliquot and store at -20C long term. Avoid freeze thaw cycles.
Isotype:IgG
Host_Name:Rabbit
Buffer:Constituents: 0.2% BSA (without Mg2+ and Ca2+), PBS. pH 7.3
ListPrice:310
AppSummary:IF
SpeciesSummary:Hu, Mu
ProteinTarget:PRAS40
PackageSize:0.1 ml
GeneralRef:Kovacina, K.S., et al. (2003) Identification of a proline-rich Akt substrate as a 14-3-3 binding partner. J. Biol. Chem. 278(12):10189-10194. Cantley, L.C. (2002) The phosphoinositide 3-kinase pathway. Science. 296(5573):1655-1657. Lizcano, J.M. and D.R. Alessi (2002) The insulin signaling pathway. Curr. Biol. 12(7):R236-R238. Cho, H., et al. (2001) Akt1/PKBalpha is required for normal growth but dispensable for maintenance of glucose homeostasis in mice. J. Biol. Chem. 276(42):38349-38352.
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